אולי כך קובעים אם לאדם יש בעית קשב
אם הוא מצליח לסיים את המבחן - הוא בסדר. (כמו התאוריה שאומרת שאם הצלחת לחשב את הסכם שלך על מנת להתקבל לאוניברסיטת ת"א - התקבלת). צירפתי את ההסברים שהיו, חשבתי שהם מיותרים. בלי ההיפראקטיביות , כי היו כאן יותר מדי תווים בשביל תפוז.. Inattentive ADD The onset of these symptoms often become apparent later in child-hood or early adolescence. The brighter the individual, the later symptoms seem to become a problem. The symptoms must be present for at least six months and not be related to a depressive episode or the onset of marijuana usage. Using marijuana can often make a person seem as though they have ADD without hyperactivity. It is important to screen for pot usage in teen-agers or adults. Girls with ADD are frequently missed because they are more likely to have the non-hyperactive form. The severity of the disorder is rated as mild, moderate or severe. Even though these children have many of the same symptoms of the people with AD/HD, they are not hyperactive and may appear hypoactive. Additional symptoms for this subtype include: excessive daydreaming, frequent complaints of being bored, appearing apathetic or unmotivated, ap-pearing frequently sluggish or slow moving or appearing spacy or internally preoccupied -- the classic "couch potato." Most people with this form of ADD are never diagnosed. They do not exhibit enough symptoms that "grate" on the environment to cause others to seek help for them. Yet, they often experience severe disability from the disorder. Instead of help, they get labeled as willful, uninterested, or defiant. As with the AD/HD subtype, brain studies in patients with ADD, inattentive subtype reveal a decrease in brain activity in the frontal lobes of the brain in response to an intellectual challenge. Again, it seems that the harder these people try to concentrate, the worse it gets. ADD, inattentive subtype is often very responsive to stimulant medications listed above, at a percentage somewhat less than the AD/HD patients . Overfocus Issues People with ADD, overfocused subtype, tend to get locked into things and they have trouble shifting their attention from thought to thought. This subtype has a very specific brain pattern, showing increased blood flow in the top, middle portion of the frontal lobes (cingulate area of the brain). This is the part of the brain that allows you to shift your attention from thing to thing. When this part of the brain is working too hard, people have trouble shifting their attention and end up "stuck" on thoughts or behaviors. This brain pattern may present itself differently among family members. For example, a mother or father with ADD overfocused subtype may experience trouble focusing, along with obsessive thoughts (repetitive negative thoughts) or compulsive behaviors (hand washing, checking, count-ing, etc.). The son or daughter may be oppositional (get stuck on saying no, no way, never, you can´t make me do it). Another family member may find change very hard for him or her. This pattern is often made worse by the stimulant medications. The problem is not inattention, but over-attention. When you give them a stimulant medication they tend to focus more on the thoughts they get stuck on. The best medications for this subtype tend to be the new "anti-obsessive antidepressants," which increase the neurotransmitter serotonin in the brain. I have nicknamed these medications "anti-stuck medications." At the time of this writing there are 9 medications which are commonly used to increase serotonin in the brain. These medications include Effexor (venlafaxine), Prozac (fluoxetine), Paxil (paroxetine), Zoloft (sertraline), Anafranil (clomi-pramine), Desyrel (trazodone), Serzone (nefazodone)), Remeron (mirtaz-apine) and Luvox (fluvoxamine). My personal favorite of these medications for overfocused ADD is Effexor. Effexor increases serotonin in the brain which is helpful for shifting attention, it also increases norepinephrine and dopamine, which are more stimulating neurotransmitters and more helpful with attentional problems. Serzone and Remeron have also been found to increase serotonin and norepinephrine. It is important to note that, in my experience, those medications which exclusively increase serotonin (Prozac, Paxil, Zoloft, Anafranil, Desyrel and Luvox) often make people with ADHD and ADD without hyperactivity worse. They tend to have more problems concentrating and they may experience decreased motivation. Limbic ADD The limbic system lies near the center of the brain. It is about the size of a walnut. This is the part of the brain that sets a person´s emotional tone or how positive or negative you are. The limbic system also affects motivation and drive. It helps get you going in the morning and encourages you to move throughout the day. It controls the sleep and appetite cycles of the body. It affects the bonding mechanism that enables you to connect with other people on a social level; your ability to do this successfully in turn influences your moods. Limbic ADD occurs when the limbic system of the brain is overactive. It may also look like depression. It is very important to differentiate between limbic ADD from clinical depression. This is best done by evaluating symptoms over time. The symptoms of limbic ADD are consistent over time, and there must have been evidence from childhood and adolescence. It does not just show up at the age of 35 when someone is going through serious stress in life. It must be a pattern of behavior over time and be associated with long term attentional difficulties. Major depressive disorders tend to cycle. There are periods of normalcy which alternate with periods of depression. The medications used for limbic ADD include standard antidepressants, such as Tofranil (imipramine), Norpramin (desipramine), and Pamelor (nortryptiline), the newer antidepressants such as Prozac (fluoxetine) and Wellbutrin (buprion), and the stimulants. Clinically, I have been very im-pressed with the ability of stimulants to help this subtype of ADD. This is one of the reasons why it is very important to differentiate this subtype from primary depressive disorders.